1. Increased body weight of offspring2. Increased fat mass, epididymal and inguinal white adipose tissue3. Increased stored lipids in fat cells 4. Increased fasting blood glucose and insulin levels5. Development of obesity and dyslipidemia6. DBP exposure may increase endoplasmic reticulum stress, which inhibits UCP1 expression, decreasing brown adipose tissue energy consumption and affecting lipid and sugar metabolism, consequently leading to obesity in the offspring. This evidence concerns the gene INS and Obesity.