MINK1 also takes part in the regulation of cell mechanical forces, which vary between metastatic and non-metastatic tumor cells, as well as between drug-resistant and drug-sensitive cells [163], which is another reason why it would require further investigation of whether MINK1 expression is altered in human neoplasms and whether the measurement of its levels could contribute to establishing more effective clinical pathways for patients. This evidence concerns the gene MINK1 and neoplasm.