To examine the progression of MCC development in the context of fxn deficiency, we conducted WISH analysis on 32 hpf animals using the trim35-30-201 and cetn2 riboprobes, which mark differentiating MCCs (Ma and Jiang, 2007; Liu et al., 2007; Marra and Wingert, 2016). This evidence concerns the gene CETN2 and Merkel cell skin cancer.