TP53 and myocardial infarction: Nevertheless, the resultsgive reason to assume that polymorphic variants rs1205*CRP,rs3732378*CX3CR1, rs1800795*IL6, rs1024611*CCL2, rs3834129*CASP8, rs1042522*TP53, rs12454712*BCL2,rs1001179*CAT, and rs10098474*MSRA significantly contributeto the formation of hereditary predisposition to MI.In addition, it was demonstrated that the identified synergisticinteractions between genotypes/alleles in combinationsof CX3CR1*A/G and CASP8*I/I, MSRA*C and CRP*C,CAT*C/T and MSRA*C, and CAT*C/T and CX3CR1*A cansignificantly affect the resulting predictive model.