ACTA1 and pulmonary fibrosis: Xie et al discovered that the glycolysis inhibitor 3PO effectively alleviated bleomycin‐induced pulmonary fibrosis and decreased the levels of collagen, fibronectin, and α‐SMA.[31] However, results from hydroxyproline immunohistochemical staining experiments conducted on mouse lung tissue sections indicated that hydroxyproline deposition persisted in the lungs of mice in the 3PO‐treated group.