Platelet‐derived growth factor BB (PDGF‐BB) and oxidized low‐density lipoprotein (ox‐LDL) are both known in vitro stimulators that mimic the pathological environment of postinjury restenosis and atherosclerosis.[6] Treatment with PDGF‐BB or ox‐LDL resulted in a dose‐dependent increase in TTK mRNA and protein levels in VSMCs (Figure 2A‐D), indicating transcriptional regulation of TTK under these conditions. Here, TTK is linked to atherosclerosis.