In their research, Ahmet et al. showed that quercetin accelerated wound healing in diabetic and non-diabetic mice through the mechanism of reducing proinflammatory cytokines [110], inhibited the secretion of cytochrome P450 2E1 (CYP2E1) during the development of diabetes, thereby preventing oxidative damage in the liver [111] and also provided neuroprotective effects through the mechanism of preventing acetylcholinesterase (AChE) activity in the brains of diabetic mice [112]. Here, ACHE is linked to diabetes mellitus.