These results indicated that the inhibition of FPR1 by Boc‐MLF led to the decrease of Cyclin D1 and NF‐ĸBp65 expression, while the activation of FPR by Ac2‐26 promoted the expression of Cyclin D1 and NF‐ĸBp65 in ovarian cancer cells. Here, FPR1 is linked to ovarian carcinoma.