In 2016, Lee et al. posited that while the precise mechanism elucidating the connection between obstetric complications and AF remains unknown, it is possible that obstetric complications, such as preeclampsia, may result in an increased adrenergic state, heightened inflammatory response, as well as activation of the renin‐angiotensin‐aldosterone system [7]. The gene discussed is REN; the disease is preeclampsia.