FDP also exert complex effects in sepsis-related liver injury, primarily through: (1) elevated levels indicating activation of the coagulation-fibrinolysis system; (2) activation of monocytes and neutrophils, leading to increased release of inflammatory factors; (3) direct damage to vascular endothelial cells; (4) induction of oxidative stress, exacerbating hepatocyte injury (30–32). This evidence concerns the gene OTOR and Sepsis.