Previous studies have shown that, by activating Wnt/β-catenin signaling pathway, transplantation of BMSC with FOXM1 overexpression can inhibit inflammation (by reducing white blood cell count, total protein concentration, inflammatory cytokines, etc.)and apoptosis, partially restore vascular integrity, thus preventing LPS-induced ARDS [13]. The gene discussed is FOXM1; the disease is acute respiratory distress syndrome.