Within this context, a previous report using GO and KEGG pathway enrichment analyses reported the potential favorable implication of myrrh in TNBC treatment via downregulation of several cancer pathways, cellular proliferation, protein bindings, apoptotic mechanism, proteoglycans in cancer, and PI3K-Akt signaling.33 In alignment with published literature, our search concluded the promising engagement of myrrh in treating various cancer types by targeting these genes’ signatures. Here, AKT1 is linked to cancer.