The clinical phenotype of anti-IgLON5 disease was initially described as bulbar dysfunction, sleep disorders (stridor, obstructive sleep apnea, REM and NREM parasomnias), abnormal movements (myoclonus, chorea, parkinsonism), gait instability, and cognitive decline,62, 66and it continues to expand. Here, IGLON5 is linked to Parasomnia.