reported that AT1001 treatment stabilized the distribution and expression of ZO‐1 within cell–cell interfaces in both the ascending and descending aortas of β‐aminopropionitrile‐fed mice.[25] In celiac disease, the barrier function is compromised, and patients with celiac disease patients exhibit enhanced intestinal permeability and disrupted TJs. The gene discussed is TJP1; the disease is celiac disease.