The precise mechanism of SARS-CoV-2-induced neurological damage remains unclear, but a possible pathway includes direct invasion into the spinal cord facilitated by the presence of angiotensin-converting enzyme 2 (ACE-2) receptors on neurons [7]. Another potential mechanism involves the cytokine storm associated with viral infections, which may result in elevated immunological factors and possibly trigger autoimmune responses [7]. This evidence concerns the gene ACE2 and viral infectious disease.