MIP1A/CCL3 and eotaxin/CCL11 are linked to AD [64, 71–73], MCP1/CCL2 levels correlate with neuroinflammation, brain atrophy, and cognitive decline in AD [74–76], and MCP3/CCL7 is part of a diagnostic panel for AD [77]. This evidence concerns the gene CCL3 and Alzheimer disease.