Another study demonstrates that IFITM1 competes with EBV-gH/gL and gB for binding to EphA2, while YTHDF3 accelerates IFITM1 mRNA degradation through m6A modification in collaboration with DDX5, thereby promoting EphA2-mediated EBV infection [195] (Fig. 5C and Table 2). The gene discussed is EPHA2; the disease is Epstein-Barr virus infection.