Overexpression of miR-29b in cholangiocarcinoma cells followed by exposure to dihydroartemisinin demonstrated increased pro-apoptotic effect compared to dihydroartemisinin alone that was associated with the concomitant upregulation of MCL-1S and downregulation of MCL-1L, however, the influence of miR-29b used alone was not assessed [41]. The gene discussed is MCL1; the disease is cholangiocarcinoma.