An integrated analysis of the transcriptomes of BC and fatty acid receptors from the public libraries targeted CXCL9 as a key player in EpOME-mediated TNBC development, which was further validated by its expression was manipulated by administration of EpOME and manipulation of CYP2J2, and its cancer-driven role in EpOME-mediated TNBC development (Fig. 5). The gene discussed is CYP2J2; the disease is cancer.