Also, malondialdehyde (MDA), carbonyl protein, tumour necrosis factor-alpha (TNF-α), interleukin-1 beta (IL-1β), nuclear factor-kappa B (NF-κB), C/EBP homologous protein (CHOP), glycogen synthase kinase-3 beta (GSK-3β), brain-derived neurotrophic factor (BDNF) and acetylcholinesterase (AChE) activity were examined.<h4>Results</h4>AzA significantly affected AlCl<sub>3</sub>-provoked anxiety-like behaviours and learning and memory impairments. This evidence concerns the gene ACHE and Anxiety.