Additionally, we observed an enrichment of immune‐related pathways in HT group (e.g., INF‐gamma/alpha‐signaling, IL6‐JAK‐STAT3‐signaling, IL2‐STAT5‐signaling, and TNFA‐signaling‐via‐NFKB, Figure 8B), suggesting that malignant cells in the HT group exhibited an inflammatory state, which might drive more interactions with the immune system after HAIC treatment. The gene discussed is IL2; the disease is hematocrit.