Interestingly, the postpartum gene expression of GLUT4 was upregulated in our study, and its level was inversely correlated with maternal HOMA-IR in the pGDM group, implying that the improvement in insulin sensitivity postpartum in the women with prior GDM could be partially due to SLC2A4 upregulation. The gene discussed is SLC2A4; the disease is gestational diabetes.