Related to this finding, a recent study by Mahe and colleagues identified a positive feedback loop between the expression of the FGFR3 receptor, a tyrosine kinase receptor strongly associated with the development and progression of BC, and the positive regulation of MYC proto-oncogene mRNA levels, thereby promoting anchorage-independent growth in bladder tumor cells [54]. This evidence concerns the gene MYC and urinary bladder neoplasm.