NFKB1 and staphylococcus aureus infection: These results emphasize that IL-17, NOD-like receptors, chemokines, Toll-like receptors, Staphylococcus aureus infection, NF-κB, JAK-STAT, and TNF, as well as chemokines and cytokines, are jointly involved in the immune and inflammatory responses induced by DNFB in atopic dermatitis mice.