DTNBP1 and schizophrenia: Finally, sandy (Sdy) mice that carry an in-frame 22-residue deletion wholly within the Dtnbp1 gene [16] exhibit a host of behavioral anomalies, including deficits in social interactions, impaired habituation, enhanced stimulant sensitization and impaired cognitive performance in fear-conditioning, novel object recognition, spatial reference, and working memory [17,18,19,20,21,22], that are consistent with a model of schizophrenia-relevant cognitive and behavioral impairment.