The modulation of pathways involving PI3K [56], Trk-A [29], Trk-B [57], Bcl2, GSK3βser9 [1], and Erk1/2 [5,58] underscores FVSEs potential role in promoting neuronal survival and function, especially in managing neurodegenerative conditions like Alzheimer’s disease [59,60]. The gene discussed is NTRK1; the disease is early-onset autosomal dominant Alzheimer disease.