Guo et al. [48] demonstrated that miR-130b-3p is overexpressed in NSCLC and that miR-130b-3p, conveyed through mesenchymal stem cell-derived extracellular vesicles, advances LC progression by inhibiting the FOXO3/NFE2L2/TXNRD1 axis. The gene discussed is FOXO3; the disease is laryngotracheoesophageal cleft.