Guo et al. [48] demonstrated that miR-130b-3p is overexpressed in NSCLC and that miR-130b-3p, conveyed through mesenchymal stem cell-derived extracellular vesicles, advances LC progression by inhibiting the FOXO3/NFE2L2/TXNRD1 axis. Here, NFE2L2 is linked to laryngotracheoesophageal cleft.