observed the overexpression of IL-17 in gastric cancer tissues through low-abundance transcriptome sequencing and concluded that IL-17 and IFN-γ are involved in the activation of T cells and B cells; moreover, IL-17 expression correlated with the activation of cytotoxic T-cell and NK cell pathways during Helicobacter pylori infection (55). The gene discussed is IL17A; the disease is gastric cancer.