KRT5 and idiopathic pulmonary fibrosis: Depending on its O‐GlcNAc status, it may switch JUNB activity across diverse environmental conditions, as it was previously observed for MORC2.[62] Intriguingly, inhibiting the O‐GlcNAc modification on JUNB attenuates the metaplastic differentiation of KRT5+ aberrant basal cells and induces regenerative features, such as the decrease of aberrant basal cells in IPF AOs.