demonstrated that specific deletion of pericytes does not alleviate renal fibrosis or the recruitment of myofibroblasts in a unilateral ureteral obstruction (UUO) mouse model.[57] However, some studies hold a different view of the potential for differentiation between pericytes and myofibroblasts in the same UUO mouse model.[58, 107] They reported that α‐SMA− interstitial pericytes differentiated into α‐SMA+ myofibroblasts during fibrosis. This evidence concerns the gene ACTA1 and renal fibrosis.