GAL and pulmonary fibrosis: In AECs, FAK1 is activated by Gal‐1 and mediates the transdifferentiation of myofibroblasts, resulting in pulmonary fibrosis.[265] Gal‐3, which is generally expressed in endothelial cells, fibroblasts, and resident alveolar macrophages, is the most studied galectin in pulmonary fibrosis caused by SARS‐CoV‐2.