We also assessed the expression of inflammatory proteins CAMP and CEBP/β, which contributed to the pathogenesis of psoriasis by amplifying inflammatory signaling pathways in keratinocytes.[13, 21] The gene expression of both CAMP and CEBPB markedly decreased to 0.7‐fold of CAMP (Figure S4C, Supporting Information) and 1.2‐fold of CEBPB (Figure S4D, Supporting Information) in the nor@MSC‐EVs group compared to 1.6‐fold and 1.4‐fold in the PBS group. The gene discussed is CEBPA; the disease is psoriasis.