While our experiments employed primary neurons instead of induced pluripotent stem cells for the hormone stimulation model, we fortuitously observed significant gene expression changes associated with depression and emotional disorders after modeling, including ERCC6L2, SLC6A4, and GAD‐1, recognized as specific biological markers for anxiety or emotional disorders.[39, 40, 41] Moreover, given the involvement of minor inflammatory responses in PTSD pathogenic mechanisms, we also investigated the expression of inflammatory factors IL‐1β, TNF‐α, and IL‐10,[42, 43] yielding positive outcomes. The gene discussed is SLC6A4; the disease is depressive symptom measurement.