Also, in pancreatic cancer, FOXD1 directly stimulates SLC2A1 transcription, suppresses SLC2A1 degradation via an RNA‐induced silencing complex, upregulates GLUT1 expression and ultimately facilitates the growth, invasion and metastasis of pancreatic cancer cells by controlling aerobic [16]. This evidence concerns the gene SLC2A1 and familial pancreatic carcinoma.