CPT1B and glioblastoma: ERRγ, upon complex formation with p65, binds to the promoter region of CPT1B and increases expression of Cpt1b which in turn is responsible for mitochondrial fatty acid oxidation (FAO) and enhanced ATP generation and oxygen consumption rate (OCR) in cancer cells.[59] FTO‐mediated m6A removal and stabilization of target mRNA transcript produces temozolomide resistance in glioblastoma.