We extended this analysis to NSCLC adenocarcinoma and SCC subtypes for mutations in epithelial growth factor receptor (EGFR), Kirsten-ras small G-protein oncogene (KRAS), serine/threonine kinase-11 (STK11) tumor suppressor genes, and Kelch-like ECH-associated protein 1 (KEAP1), a redox-sensitive transcriptional modulator (Fig. S1). This evidence concerns the gene STK11 and non-small cell lung carcinoma.