NR2F2 and influenza: Meanwhile, after influenza injury, nuclear factor κB inhibits COUP‐TFII, but surviving ECs ultimately rely on recovery of COUP‐TFII to reestablish vascular homeostasis.[60] These previous studies supported the implementation of COUP‐TFII over‐expression as a strategy to promote endothelialization and mitigate risk of adverse remodeling occurrence in our developed decellularized allografts.