They in fact analyzed a regulatory mechanism between the MALAT1 and NRXN1 through the interaction of two microRNAs, miR-141-3p and 200a-3p, establishing a so-called “MALAT1-miR-141-3p/200a-3p-NRXN1” axis, which impairs the synthesis of several synapse-related proteins in ADHD and could be used as diagnostic biomarkers. This evidence concerns the gene NRXN1 and attention deficit-hyperactivity disorder.