The original tumor cells were positive for 5-HT, substance P, VMAT1/2, and markers for neuroendocrine phenotype, namely CgA, SYP, SV2, and NCAM, via IHC; the original tumor and the derived cell line also expressed somatostatin receptors, including SSTR2, which are the main treatment targets for these malignancies. This evidence concerns the gene TAC1 and neoplasm.