To confirm that hemocytes are the source of the Egr signal activating JNK in the tumor, we specifically knocked down egr in hemocytes (Hml-Gal4) or the fat body (R4-Gal4, Lpp-Gal4) while inducing tumor formation with retn-lexA–driven lexAop-NICD. Here, CD1A is linked to neoplasm.