We believe thatrelatively high liver SUVmax may lead to missed lesions (16,17).Notably, PPGLs are a unique type of tumor that differs from other neuroendocrinetumors of epithelial origin and exhibit substantially higher somatostatin receptor 2(SSTR2) expression than normal tissues (7).Functionally, SSTR2 can mediate the release of somatostatin, thereby inhibitingendothelial cell proliferation and inducing cell apoptosis. Here, SST is linked to neoplasm.