We found that the single known risk variant for COVID-related anosmia was significant in the “ability to smell” GWAS data, but the effect size was small (UGT2A1/UGT2A2 locus rs7688383, BetaAbility to smell = 0.0077, p = 1.2 × 10−11; ORCOVID-19 anosmia = 1.11, p = 1.4 × 10−14)19. This evidence concerns the gene UGT2A2 and Kallmann syndrome.