It has been shown that c-MET-dependent nitric oxide release by neutrophils effectively inhibited tumor growth and metastasis, indicating the pivotal role of MET in recruiting anti-tumor neutrophils.365 However, a subsequent study revealed that inhibiting c-MET can hinder the reactive recruitment of neutrophils from the bone marrow into tumor tissues, facilitating T cell infiltration into tumors, and potentially improving the efficacy of cancer immunotherapy.366 These divergent findings highlight a dual role for c-MET in neutrophil immune responses within the tumor microenvironment. Here, MET is linked to cancer.