Specifically, elevated expression of SCGB1A1 and ZYX during hAHI and decreased levels of LILRA3 were associated with the absence of ARS; increased levels of NAPA and RAN during hAHI, and decreased levels of ITIH4 after hAHI were associated with lower viremia; and increased levels of HPN and PRKCB during hAHI, and ITGB3 and DDTL after hAHI, were associated with faster disease progression. The gene discussed is HPN; the disease is Axenfeld-Rieger syndrome.