GSK3B and neoplasm: In Mia PaCa‐2 cells, the suppression of GSK‐3β inhibits mutant KRas‐dependent tumor growth and the depletion of GSK‐3β increases β‐catenin levels and forces the expression of β‐catenin mutants that lack GSK3 phosphorylation sites and are thus insufficient to induce apoptosis.