Analysis of kidney biopsy samples from 28 DKD patients revealed a decrease in the expression of mRNAs for both glutathione peroxidase 4 (GPX4) and solute carrier family 7 member 11 (SLC7A11) in the kidney tubules of DKD patients compared to those in healthy individuals.[8] GPX4 and SLC7A11 are critical modulators of ferroptotic cell death,[9] and thus we postulated that ferroptosis might play a role in the renal protective effects mediated by GLP‐1RAs. This evidence concerns the gene SLC17A1 and diabetic kidney disease.