Similarly, the levels of renal tubular injury markers N‐acetyl‐β‐glucosaminidase (NAG), transferrin, kidney injury molecule 1 (KIM‐1), and neutrophil gelatinase‐associated lipocalin (NGAL) were significantly increased in the DKD group, whereas their expression was markedly repressed under Smg treatment and ferroptosis inhibition (Figure 2L; Figure S2B–D, Supporting Information). This evidence concerns the gene LCN2 and diabetic kidney disease.