Furthermore, the main focus of Flt3L research has been its potential to generate DCs from bone marrow progenitors, with a limited understanding of how these cells influence CD8+ T‐cell niches.[45] Unraveling the molecular mechanisms by which DCs preserve niches for CD8+ T cells will offer novel immunotherapy targets for reshaping the TME of NSCLC through the integration of MWA. The gene discussed is FLT3LG; the disease is non-small cell lung carcinoma.