Tumor growth was suppressed compared to the non-treated group in WT → IFNγR1flox/flox mice (Figures 2C-D) but no suppression was observed in WT → Tie2CreIFNγR1flox/flox mice, suggesting that IFN-γR1 expression in endothelial cells was indispensable for the success of ACT. This evidence concerns the gene IFNGR1 and neoplasm.