To evaluate the potential role of GATA2 in the pathogenesis of HBV-related HCC, we created siRNA molecules and overexpression plasmids targeting GATA2, and our data demonstrated that these constructs significantly impacted KIF20A expression levels in HepG2.2.15 cells (p < 0.001, Figures 7A, B). Here, KIF20A is linked to hepatocellular carcinoma.