TNF and myocardial infarction: Swiftly infiltratingLy6Chigh monocytes evolve into M1 macrophages, which promote inflammation byengulfing cell debris, breaking down extracellular matrix (ECM), and secretingpro-inflammatory factors (such as tumor necrosis factor alpha (TNF-α),interleukin-1 beta (IL-1β), and interleukin-6 (IL-6)), thus triggering therepair processes after myocardial infarction.