MiR-124-3p was down-regulated in LAM tissues of patients (n = 4; p < 0.05) and inhibited the apoptosis of TSC2-deficient cells through the oxidation of peroxisomal fatty acids mediated by retinoid X receptor α (RXRα) [89], indicating that RXRα might be the target of miR-124 in LAM. The gene discussed is RXRA; the disease is lymphangioleiomyomatosis.